TYPE B AORTIC INTRAMURAL HEMATOMA – WHEN A SHEEP BECOMES A WOLF

Authors

  • Nuno Henriques Coelho Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Clara Nogueira Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Victor Martins Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Jacinta Campos Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Andreia Coelho Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Rita Augusto Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Evelise Pinto Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Carolina Semião Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • João Ribeiro Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • João Peixoto Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal
  • Alexandra Canedo Serviço de Angiologia e Cirurgia Vascular, Centro Hospitalar Vila Nova de Gaia/Espinho, Portugal

DOI:

https://doi.org/10.48750/acv.245

Keywords:

Acute aortic syndromes, Intramural hematoma, Penetrating aortic ulcer, TEVAR

Abstract

Introduction: Type B aortic intramural hematoma (B-IMH) has a complex and variable natural history: it can remain stable and resolve spontaneously or progress to dissection, aneurysm, ulcer or even rupture. The possibility of disease progression, frequently with mild or no symptomatology, poses a significant treatment dilemma.


Clinical Case: We report a case of a 60 year-old-female diagnosed with an acute B-IMH, initially treated medically. However, 1-month control CTA revealed disease progression (increased B-IMH thickness and evolution to an ulcer-like-projection with 20 mm diameter and 11 mm depth). She was submitted to a left carotid-subclavian bypass followed by TEVAR and left-subclavian ostial embolization. During follow-up (5 months) patient remain asymptomatic, demonstrating favorable aortic remodeling.


Conclusion: Type B-IMH is a dynamic pathology. From presentation to late follow-up, patients remain at high risk for abrupt catastrophic complications. As reported, TEVAR seems to be a safe and effective approach in the event of unfavorable evolution.

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References

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Published

2020-02-07

How to Cite

1.
Coelho NH, Nogueira C, Martins V, Campos J, Coelho A, Augusto R, Pinto E, Semião C, Ribeiro J, Peixoto J, Canedo A. TYPE B AORTIC INTRAMURAL HEMATOMA – WHEN A SHEEP BECOMES A WOLF. Angiol Cir Vasc [Internet]. 2020 Feb. 7 [cited 2024 Nov. 23];15(4):269-72. Available from: https://acvjournal.com/index.php/acv/article/view/245

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Section

Clinical Case